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- W2023458100 abstract "Background: Laparoscopic liver resection is a relatively new surgical procedure. Carbon dioxide (CO 2 ) pneumoperitoneum and laparoscopic liver dissection are recognized as risk factors for CO 2 embolism to the pulmonary circulation. The embolization can be difficult to detect and can theoretically increase peri‐operative morbidity. The aim of this study was to evaluate the cardiopulmonary effects in a pig model during a time period of 4 h after an experimental CO 2 embolization. Methods: Eleven piglets were anesthesized. Nine were embolized with a single intravenous injection of 0.4 ml/kg CO 2 and two served as controls. Respiratory and cardiovascular variables, including pulmonary artery pressure and cardiac output, were monitored for 4 h after embolization, and arterial blood gases were monitored on‐line. Results: The embolized piglets had an increase in ventilatory dead space, pulmonary vascular resistance and pulmonary artery pressure and a decrease in cardiac output that lasted throughout the 4‐h observation time. The mean arterial pressure and heart rate were unchanged. An early sign of embolization was a rapid fall in end‐tidal CO 2 and P a o 2 and a rise in P a co 2 . Conclusion: Negative changes in cardiopulmonary physiology persisted for at least 4 h after a single intravenous CO 2 injection, in spite of this gas being highly soluble in blood. This is a more prolonged influence of CO 2 embolization than previously described. Extensive monitoring for early detection of an embolization may be recommended to limit morbidity in patients undergoing laparoscopic liver surgery." @default.
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- W2023458100 date "2006-01-18" @default.
- W2023458100 modified "2023-10-12" @default.
- W2023458100 title "The effects of experimental venous carbon dioxide embolization on hemodynamic and respiratory variables" @default.
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- W2023458100 doi "https://doi.org/10.1111/j.1399-6576.2006.00933.x" @default.
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