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- W2023616904 endingPage "1557" @default.
- W2023616904 startingPage "1547" @default.
- W2023616904 abstract "HPVs are the causative agents of cervical and other anogenital cancers. HPVs infect stratified epithelia and link their productive life cycles to cellular differentiation. Low levels of viral genomes are stably maintained in undifferentiated cells and productive replication or amplification is restricted to differentiated suprabasal cells. Amplification is dependent on the activation of the ATM DNA damage factors that are recruited to viral replication centers and inhibition of this pathway blocks productive replication. The STAT-5 protein appears to play a critical role in mediating activation of the ATM pathway in HPV-positive cells. While HPVs need to activate the DNA damage pathway for replication, cervical cancers contain many genomic alterations suggesting that this pathway is circumvented during progression to malignancy." @default.
- W2023616904 created "2016-06-24" @default.
- W2023616904 creator A5007196678 @default.
- W2023616904 creator A5023408072 @default.
- W2023616904 date "2013-12-01" @default.
- W2023616904 modified "2023-10-10" @default.
- W2023616904 title "Regulation of the life cycle of HPVs by differentiation and the DNA damage response" @default.
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