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• W2023643147 abstract "Fast responses to extracellular ATP are mediated by the activation of P2X receptors. Native and cloned P2X receptors are permeable to monovalent cations such as Na+ and K+ as well as divalent cations such as Ca2+. However, altered P2X receptor expression has not been definitively determined under pathological conditions, particularly in epilepsy. Here we show that, in the seizure-sensitive (SS) gerbil hippocampus, a recognized genetic epilepsy model, the expressions of both P2X2 and P2X4 receptors are markedly decreased as compared with that in the seizure-resistant (SR) gerbil. These alterations are closely related to changes in gamma-aminobutyric acid (GABA) concentrations induced by vigabatrin (VGB) or 3-mercaptopropionic acid (3-MPA) treatment. Furthermore, the regulation of both P2X receptor expression in the gerbil hippocampus was mediated by the GABA(A) receptor, not GABA(B). These results suggest that the GABA(A) receptor-mediated modulation of P2X receptor expression may play an important role in the regulation of neuronal excitability." @default.
• W2023643147 created "2016-06-24" @default.
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• W2023643147 date "2003-08-01" @default.
• W2023643147 modified "2023-10-06" @default.
• W2023643147 title "P2X2 and P2X4 receptor expression is regulated by a GABAA receptor-mediated mechanism in the gerbil hippocampus" @default.
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• W2023643147 doi "https://doi.org/10.1016/s0169-328x(03)00260-2" @default.
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