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• W2023751947 endingPage "249" @default.
• W2023751947 startingPage "241" @default.
• W2023751947 abstract "Neuroprotective strategies can prevent lesions from getting worse but agents that have neurotrophic properties can also affect repair in a developing brain. Although prevention and treatment in the early stages of brain lesions are desirable, delayed cell death or improved post-lesion plasticity are the only realistic targets in many cases. Several trophic factors can limit delayed cell death in animal models of perinatal brain damage. In addition, melatonin and brain-derived neurotrophic factor have been shown to promote post-lesion plasticity following neonatal excitotoxic white-matter damage in newborn mice. Despite these promising results, additional preclinical data are required for most of the trophic factors that have been tested, although some candidate drugs, e.g. melatonin or erythropoietin, might reach clinical trials in the near future." @default.
• W2023751947 created "2016-06-24" @default.
• W2023751947 creator A5033474449 @default.
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• W2023751947 date "2007-08-01" @default.
• W2023751947 modified "2023-10-12" @default.
• W2023751947 title "Growth factors and plasticity" @default.
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• W2023751947 doi "https://doi.org/10.1016/j.siny.2007.01.007" @default.
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