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• W2023757802 endingPage "911" @default.
• W2023757802 startingPage "899" @default.
• W2023757802 abstract "Extracellular K⁺, Na⁺, and Ca²⁺ ions all influence the resting membrane potential of the neuron. However, the mechanisms by which extracellular Na⁺ and Ca²⁺ regulate basal neuronal excitability are not well understood. Recent findings suggest that NALCN, in association with UNC79 and UNC80, contributes a basal Na⁺ leak conductance in neurons. Mutations in Nalcn, Unc79, or Unc80 lead to severe phenotypes that include neonatal lethality and disruption in rhythmic behaviors. This review discusses the properties of the NALCN complex, its regulation, and its contribution to neuronal function and animal behavior." @default.
• W2023757802 created "2016-06-24" @default.
• W2023757802 creator A5010578085 @default.
• W2023757802 date "2011-12-01" @default.
• W2023757802 modified "2023-10-14" @default.
• W2023757802 title "Sodium Leak Channels in Neuronal Excitability and Rhythmic Behaviors" @default.
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