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- W2023843953 endingPage "343" @default.
- W2023843953 startingPage "335" @default.
- W2023843953 abstract "Tuberculosis is a global health calamity. The causative agent, Mycobacterium tuberculosis (M. tuberculosis), has evolved elaborate survival mechanisms in humans, allowing it to remain in a clinically latent infection state, constantly engaging the immune system, with the possibility to progress to active disease. Autophagy is a cellular process responsible for the degradation of intracellular components, including invading pathogens, playing an important role in both innate and adaptive immunity. In this review, we describe the molecular mechanisms employed by M. tuberculosis to avoid autophagic degradation and exploit this process to its own advantage. Moreover, we discuss the multiple roles played by autophagy in the immune responses to M. tuberculosis, and its unforeseen contribution to the antibacterial activity of tuberculosis-specific drugs." @default.
- W2023843953 created "2016-06-24" @default.
- W2023843953 creator A5000329723 @default.
- W2023843953 creator A5046139662 @default.
- W2023843953 creator A5050710377 @default.
- W2023843953 creator A5055234275 @default.
- W2023843953 creator A5058381480 @default.
- W2023843953 date "2013-08-01" @default.
- W2023843953 modified "2023-09-25" @default.
- W2023843953 title "Autophagy in Mycobacterium tuberculosis infection: A passepartout to flush the intruder out?" @default.
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