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• W2023921534 abstract "Passafaro D, Reina S, Sterin‐Borda L, Borda E. Cholinergic autoantibodies from primary Sjögren’s syndrome modulate submandibular gland Na + /K + ‐ATPase activity via prostaglandin E 2 and cyclic AMP. Eur J Oral Sci 2010; 118: 131–138. © 2010 The Authors. Journal compilation © 2010 Eur J Oral Sci We demonstrate that patients with primary Sjögren’s syndrome (pSS) produce functional IgG autoantibodies that interact with the glandular M 3 muscarinic acetylcholine receptors (mAChRs). These autoantibodies act as a partial muscarinic agonist, increasing prostaglandin E 2 (PGE 2 ) and cyclic AMP production through modifying Na + /K + ‐ATPase activity, but also interfere with the secretory effect of the parasympathetic neurotransmitter. The IgG from patients with pSS has two effects on the submandibular gland. On the one hand, it may act as an inducer of the proinflammatory molecule (PGE 2 ) that, in turn, inhibits Na + /K + ‐ATPase activity. On the other hand, it plays a role in the pathogenesis of dry mouth, abolishing the Na + /K + ‐ATPase inhibition and the net K + efflux stimulation of the salivary gland in response to the authentic agonist pilocarpine, decreasing salivary fluid production." @default.
• W2023921534 created "2016-06-24" @default.
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• W2023921534 date "2010-03-11" @default.
• W2023921534 modified "2023-10-16" @default.
• W2023921534 title "Cholinergic autoantibodies from primary Sjögren’s syndrome modulate submandibular gland Na⁺/K⁺-ATPase activity via prostaglandin E₂and cyclic AMP" @default.
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• W2023921534 doi "https://doi.org/10.1111/j.1600-0722.2010.00716.x" @default.
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