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- W2023985059 endingPage "123" @default.
- W2023985059 startingPage "103" @default.
- W2023985059 abstract "Bacterial persistence is caused by the presence of rare, slowly growing bacteria among populations of rapidly growing cells. The slowly growing bacteria are tolerant of antibiotics and other environmental insults, whereas their isogenic, rapidly growing siblings are sensitive. Recent research has shown that persistence of the model organism Escherichia coli depends on toxin-antitoxin (TA) loci. Deletion of type II TA loci reduces the level of persistence significantly. Lon protease but no other known ATP-dependent proteases is required for persistence. Polyphosphate and (p)ppGpp also are required for persistence. These observations led to the proposal of a simple and testable model that explains the persistence of E. coli. It is now important to challenge this model and to test whether the persistence of pathogenic bacteria also depends on TA loci." @default.
- W2023985059 created "2016-06-24" @default.
- W2023985059 creator A5047204599 @default.
- W2023985059 creator A5070618817 @default.
- W2023985059 date "2012-10-13" @default.
- W2023985059 modified "2023-10-16" @default.
- W2023985059 title "Bacterial Persistence and Toxin-Antitoxin Loci" @default.
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