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- W2023991572 abstract "Mitochondrial Ca2+ sequestration likely contributes to cell death in excitotoxicity and ischemia reperfusion injury, and may also be involved in chronic forms of neurodegeneration in which a compromise in bioenergetic function alters cellular Ca2+ homeostasis. Bcl-2 overexpression is known to protect against Ca(2+)-mediated death; the mechanism of protection remains unresolved. Our data of the ability of Bcl-2 to potentiate mitochondrial Ca2+ uptake capacity and resistance to Ca(2+)-induced damage is discussed in light of current information on apoptotic signaling pathways." @default.
- W2023991572 created "2016-06-24" @default.
- W2023991572 creator A5013957802 @default.
- W2023991572 date "1999-11-01" @default.
- W2023991572 modified "2023-09-23" @default.
- W2023991572 title "Ca2+-Mediated Mitochondrial Dysfunction and the Protective Effects of Bcl-2" @default.
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- W2023991572 doi "https://doi.org/10.1111/j.1749-6632.1999.tb07815.x" @default.
- W2023991572 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/10672227" @default.
- W2023991572 hasPublicationYear "1999" @default.
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