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- W2024070385 endingPage "203" @default.
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- W2024070385 abstract "Inflammation underpins the development of atherosclerosis. Initiation and progression of vascular inflammation involves a complex cellular network, with macrophages as major contributors. Activated macrophages produce proinflammatory mediators, bridge innate and adaptive immunity, regulate lipid retention, and participate directly in vascular repair and remodeling. Recent efforts to elucidate molecular mechanisms involved in the regulation of vascular inflammation in atherosclerosis have implicated several families of innate immune recognition receptors in inflammatory activation during the course of this disease. This article reviews our current understanding of innate immune recognition receptors, signaling pathways, and putative ligands implicated in activation of macrophages in the disease. In its final section, we propose a model for the role of macrophages in bridging inflammation and atherosclerosis from the perspective of innate immune recognition and activation." @default.
- W2024070385 created "2016-06-24" @default.
- W2024070385 creator A5003021166 @default.
- W2024070385 creator A5004855576 @default.
- W2024070385 date "2007-09-10" @default.
- W2024070385 modified "2023-10-17" @default.
- W2024070385 title "Innate immunity, macrophage activation, and atherosclerosis" @default.
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