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- W2024235550 endingPage "815" @default.
- W2024235550 startingPage "802" @default.
- W2024235550 abstract "Alterations in oxidative phosphorylation resulting from mitochondrial dysfunction have long been hypothesized to be involved in tumorigenesis. Mitochondria have recently been shown to play an important role in regulating both programmed cell death and cell proliferation. Furthermore, mitochondrial DNA (mtDNA) mutations have been found in various cancer cells. However, the role of these mtDNA mutations in tumorigenesis remains largely unknown. This review focuses on basic mitochondrial genetics, mtDNA mutations and consequential mitochondrial dysfunction associated with cancer. The potential molecular mechanisms, mediating the pathogenesis from mtDNA mutations and mitochondrial dysfunction to tumorigenesis are also discussed." @default.
- W2024235550 created "2016-06-24" @default.
- W2024235550 creator A5001221849 @default.
- W2024235550 creator A5024986601 @default.
- W2024235550 creator A5076621889 @default.
- W2024235550 date "2009-06-16" @default.
- W2024235550 modified "2023-10-14" @default.
- W2024235550 title "Implications of mitochondrial DNA mutations and mitochondrial dysfunction in tumorigenesis" @default.
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