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• W2024343622 abstract "In guinea-pig ileal longitudinal smooth muscle, both palytoxin (PTX) and carbachol (CCh) increased K+ efflux with an EC50 of 1.8×10−10 M and 4.1 ×10−7 M, respectively. Atropine (10−6 M) did not inhibit the K+ efflux due to PTX (3×10−9 M), but completely inhibited the efflux due to CCh (1Cr5 M). External Ca2+ removal and verapamil (10−5 M) did not change the PTX-induced K+ efflux, although the CCh-induced K+ efflux was inhibited about 77% and 71%, respectively. PTX-induced K+ efflux was reduced to 31% by a depletion of intracellular Ca2+. Tetraethylammonium (15 mM) inhibited the K+ efflux due to PTX or CCh to 61% or 75%, respectively. The PTX-induced K+ efflux was also inhibited by cymarin (3×10−8 M), ouabain (10−5 M) and digitoxin (10−5 M). These results suggest that the PTX-induced K+ efflux is less dependent on Ca2+ influx than that due to CCh. Furthermore, the binding sites for PTX in the ileal muscle of guinea-pig may be Na+,K+-ATPase, as has been suggested in other types of cells." @default.
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• W2024343622 date "1988-01-01" @default.
• W2024343622 modified "2023-09-23" @default.
• W2024343622 title "Palytoxin-induced K+ efflux from ileal longitudinal smooth muscle of the guinea-pig." @default.
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• W2024343622 doi "https://doi.org/10.1254/jjp.46.285" @default.
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