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- W2024407826 endingPage "2206" @default.
- W2024407826 startingPage "2198" @default.
- W2024407826 abstract "Brain cells in Alzheimer's disease (AD) exhibit a membrane defect characterized by accelerated phospholipid turnover. The mechanism responsible for this defect remains unknown. Recent studies indicate that impairment of mitochondrial function is frequently observed in AD and may be responsible for certain aspects of its pathophysiology. We show that when PC12 cells are exposed to inhibitors of mitochondrial bioenergetics, the turnover of their major membrane phospholipid, phosphatidylcholine, is accelerated, producing a pattern of metabolic changes that mimics that observed in brains of AD patients. Abnormalities of mitochondrial function may therefore underlie the membrane defect in AD." @default.
- W2024407826 created "2016-06-24" @default.
- W2024407826 creator A5039868907 @default.
- W2024407826 creator A5043095398 @default.
- W2024407826 creator A5086674512 @default.
- W2024407826 date "2000-11-01" @default.
- W2024407826 modified "2023-10-15" @default.
- W2024407826 title "Role of single‐stranded DNA regions and Y‐box proteins in transcriptional regulation of viral and cellular genes" @default.
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- W2024407826 doi "https://doi.org/10.1096/fj.99-0853" @default.
- W2024407826 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11053240" @default.
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