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- W2024486446 endingPage "133" @default.
- W2024486446 startingPage "110" @default.
- W2024486446 abstract "Bipolar disorder (BPD) is characterized by recurrent episodes of disturbed affect including mania and depression as well as changes in psychovegetative function, cognitive performance, and general health. A growing body of data suggests that BPD arises from abnormalities in synaptic and neuronal plasticity cascades, leading to aberrant information processing in critical synapses and circuits. Thus, these illnesses can best be conceptualized as genetically influenced disorders of synapses and circuits rather than simply as deficits or excesses in individual neurotransmitters. In addition, commonly used mood-stabilizing drugs that are effective in treating BPD have been shown to target intracellular signaling pathways that control synaptic plasticity and cellular resilience. In this article we draw on clinical, preclinical, neuroimaging, and post-mortem data to discuss the neurobiology of BPD within a conceptual framework while highlighting the role of neuroplasticity in the pathophysiology and treatment of this disorder." @default.
- W2024486446 created "2016-06-24" @default.
- W2024486446 creator A5047437809 @default.
- W2024486446 creator A5057147306 @default.
- W2024486446 creator A5068876839 @default.
- W2024486446 creator A5071982754 @default.
- W2024486446 date "2007-10-03" @default.
- W2024486446 modified "2023-09-30" @default.
- W2024486446 title "Cellular Plasticity Cascades in the Pathophysiology and Treatment of Bipolar Disorder" @default.
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