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• W2024493338 startingPage "1089" @default.
• W2024493338 abstract "Abstract Arabidopsis plants containing the ndr1-1 mutation are incapable of mounting a hypersensitive response to bacteria carrying avrRpt2, but show an exaggerated cell death response to bacteria carrying avrB (Century et al., 1995). We show here that ndr1-1 plants are severely impaired in induction of systemic acquired resistance andPR1-driven transcription of a reporter gene in response to Pseudomonas syringae strains carryingavrRpt2 but not in response to P. syringae carrying avrB. Thendr1-1 mutation also impaired salicylic acid (SA) accumulation in response to treatments that produced reactive oxygen species (ROS) and impaired induction of systemic acquired resistance in response to in situ production of ROS. Hydrogen peroxide accumulated in wild-type Arabidopsis leaves beginning 4 to 7 h postinoculation with P. syringae carrying either avrRpt2or avrB. In ndr1-1 plants, P. syringae carrying avrRpt2 elicited no detectable hydrogen peroxide production. Hydrogen peroxide production in response to bacteria carrying avrB was similar to that of Columbia in kinetics but of lesser intensity at early time points. These data are interpreted to indicate that NDR1 links ROS generation to SA production and that the phenotypic consequences of the ndr1-1 mutation are caused by a reduced ability to accumulate SA upon pathogen infection." @default.
• W2024493338 created "2016-06-24" @default.
• W2024493338 creator A5066054326 @default.
• W2024493338 creator A5067226371 @default.
• W2024493338 date "2001-11-01" @default.
• W2024493338 modified "2023-10-11" @default.
• W2024493338 title "The Role of<i>NDR1</i>in Avirulence Gene-Directed Signaling and Control of Programmed Cell Death in Arabidopsis" @default.
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• W2024493338 doi "https://doi.org/10.1104/pp.010096" @default.
• W2024493338 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/129278" @default.
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• W2024493338 hasPublicationYear "2001" @default.
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