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• W2024604197 abstract "Human cells can process DNA double-strand breaks (DSBs) by either homology directed or non-homologous repair pathways. Defects in components of DSB repair pathways are associated with a predisposition to cancer. The products of the BRCA1 and BRCA2 genes, which normally confer protection against breast cancer, are involved in homology-directed DSB repair. Defects in another homology-directed pathway, single-strand annealing, are associated with genome instability and cancer predisposition in the Nijmegen breakage syndrome and a radiation-sensitive ataxia-telangiectasia-like syndrome. Many DSB repair proteins also participate in the signaling pathways which underlie the cell's response to DSBs." @default.
• W2024604197 created "2016-06-24" @default.
• W2024604197 creator A5035270156 @default.
• W2024604197 date "2000-04-01" @default.
• W2024604197 modified "2023-09-27" @default.
• W2024604197 title "DNA double strand break repair in mammalian cells" @default.
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• W2024604197 doi "https://doi.org/10.1016/s0959-437x(00)00069-1" @default.
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