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- W2024604828 abstract "The p53 tumour suppressor protein can efficiently inhibit tumour development. This activity reflects its ability to induce a number of different responses, including cell cycle arrest and apoptosis. Recent studies have revealed some interesting insights into how the choice of response to p53 is regulated, highlighting a correlation between the activation of cell cycle arrest and survival with the ability of p53 to reduce oxidative stress and protect cells from genotoxic damage. Understanding the molecular mechanisms that determine which response is selected may allow us to modulate these pathways so that therapeutic reactivation of p53 favours apoptotic cell death in tumour cells, but a reversible - and therefore far less toxic - induction of cell cycle arrest in normal cells." @default.
- W2024604828 created "2016-06-24" @default.
- W2024604828 creator A5090116969 @default.
- W2024604828 date "2006-12-15" @default.
- W2024604828 modified "2023-10-02" @default.
- W2024604828 title "Outcomes of p53 activation - spoilt for choice" @default.
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- W2024604828 doi "https://doi.org/10.1242/jcs.03293" @default.
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