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- W2024889734 abstract "The contribution to neonatal pulmonary injury by reactive oxygen species has been difficult to assess. The recent discovery of a novel class of prostaglandins, the isoprostanes, may help as these substances appear to be generated in response to lipid peroxidation. Isoprostanes are noncylooxygenase derived and are produced in vivo. They may contribute to pulmonary pathophysiology by their vasoconstrictive properties. We determined if 8-epi-PGF2α, a member of the isoprostane family, would be detectable in the urine or tracheal aspirates of infants ≥ 37 weeks GA with pulmonary disorders treated with assisted ventilation and FiO2 of 1.0 and in control infants without pulmonary disorders, breathing room air. Isoprostane levels were measured by enzyme immune assay (EIA). Urine levels were normalized by the urine creatinine and tracheal aspirate levels by free Secretory Component (fSC) of IgA, also measured by EIA. Results are as follows: Table The 24 and 48 hr tracheal aspirate values in sick infants were not different. When sick and control groups combined data were compared, there was a significant difference (p<0.02). No differences were found in urine values. Serial urine samples were obtained from a separate group of 15 term healthy infants to analyze isoprostane levels at different age periods. Table" @default.
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- W2024889734 date "1996-04-01" @default.
- W2024889734 modified "2023-10-18" @default.
- W2024889734 title "ISOPROSTANES IN NEONATAL PULMONARY DISEASES: POSSIBLE MARKER OF LUNG INJURY. † 1980" @default.
- W2024889734 doi "https://doi.org/10.1203/00006450-199604001-02004" @default.
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