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- W2025705938 abstract "Subchronic treatment with the β2-adrenoceptor agonist, clenbuterol (0.3 mg · kg−1, twice daily for 14 days), significantly increased the median blood pressure in anaesthetized normotensive rats. The treatment produced a marked reduction in the vasodilator effect of isoproterenol. Acute clenbuterol administration (0.01 mg·kg− i.v.) reduced the contractile response induced by the α2-adrenoceptor agonists, guanabenz or B-HT 920, and the α1- and α2-adrenoceptor agonist, clonidine, whereas it did not affect the vasoconstriction induced by the α1-adrenoceptor agonist, methoxamine, in the pithed rat. Subchronic treatment with clenbuterol attenuated the effect of the β2-adrenoceptor agonist on the vascular α2-adrenoceptor responses and enhanced the α1-adrenoceptor response to phenylephrine in pithed rats. The effect of the β2-adrenoceptor agonist was reduced by 1- but not d-propranolol. These results suggest that subchronic treatment with clenbuterol produces a subsensitivity of the β2-adrenoceptors and reduces the interaction between β2- and α2-adrenoceptors at the vascular wall." @default.
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- W2025705938 title "Interaction between β2- and α2-adrenoceptor responses in the vascular system: effect of clenbuterol" @default.
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- W2025705938 doi "https://doi.org/10.1016/0014-2999(86)90190-1" @default.
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