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- W2025785025 endingPage "579" @default.
- W2025785025 startingPage "571" @default.
- W2025785025 abstract "Among the host defense mechanisms against bacteria, leukocyte phagocytosis leads to their hydrogen peroxide (H(2)O(2))-mediated destruction. The recent discovery of dual oxidase (DUOX)-dependent H(2)O(2) generation associated with peroxidase and thiocyanate secretion at the apex of mucosal cells has been similarly interpreted as a killing mechanism. However, the rapid degradation of H(2)O(2) would be expected to reduce the efficiency of this system. It has been demonstrated that H(2)O(2) acts as a chemorepellent for bacteria, and such an effect might be sufficient to block cellular infection. Therefore, H(2)O(2) generation might represent one of the mechanisms that allows the coexistence of mucosae with potentially harmful bacteria. Here, we discuss the possible role of DUOXes and H(2)O(2) in interactions between host mucosae and bacteria to maintain mucosal homeostasis." @default.
- W2025785025 created "2016-06-24" @default.
- W2025785025 creator A5035311400 @default.
- W2025785025 creator A5036202127 @default.
- W2025785025 creator A5054560560 @default.
- W2025785025 creator A5069915743 @default.
- W2025785025 creator A5091101919 @default.
- W2025785025 date "2009-12-01" @default.
- W2025785025 modified "2023-10-02" @default.
- W2025785025 title "Dual oxidases and hydrogen peroxide in a complex dialogue between host mucosae and bacteria" @default.
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