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- W2026674202 abstract "Rothmund Thomson syndrome (RTS) is an oculocutaneous and cancer-prone disorder in which enehanced carcinogen sensitivity, mediated through abnormal DNA metabolism, may be an associated factor. Cultured fibroblasts from 4 RTS patients have been examined for their colony-forming abilities and DNA repair capacities following γ-irradiation. 2 of the 4 RTS strains showed enhanced sensitivity following hypoxic γ-radiation, and 1 of these 2 strains also showed enhanced sensitivity under oxic conditions. Defective DNA repair was implicated in the above abnormal responses to γ-radiation since both strains displayed reduced levels of repair synthesis and slow removal of radiogenci DNA lesions (assayed by their sensitivity to strand-incising activities present in protein extracts of Micrococcus leteus cells). A hypothesis is presented to rationalize the origin and heterogeneity of these laboratory phenotypes of RTS." @default.
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- W2026674202 date "1982-05-01" @default.
- W2026674202 modified "2023-10-01" @default.
- W2026674202 title "Enhanced radiosensitivity and defective DNA repair in cultured fibroblasts derived from Rothmund Thomson syndrome patients" @default.
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- W2026674202 doi "https://doi.org/10.1016/0027-5107(82)90183-x" @default.
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