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• W2027166013 abstract "Modulation of vasodilating actions by quercetin, a kind of flavonoid, was investigated using rat mesenteric arterial ring strips.Ring strips (1mm) of rat mesenteric artery were used. The specimens were kept at 36.5 °C in Krebs-Henseleit solution oxygenated with 95% O(2) and 5% CO(2).Quercetin (0.1 to 100 μM) dilated the contraction induced by norepinephrine (1 μM) in a concentration-dependent manner. The quercetin-induced vasodilatation was almost resistant to both 100 μM L-N(G)-nitro arginine methyl ester (L-NAME) and 100 μM indomethacin. At 1mM tetraethylammonium (a KCa channel inhibitor) decreased the quercetin-induced vasodilatation, which was resistant to L-NAME and indomethacin, but not significantly. L-NAME- and indomethacin-resistant quercetin-induced vasodilatation was significantly attenuated by 100 μM 18α- and 50 μM 18β-glycyrrhetinic acids (gap junction inhibitors). Endothelium removal as well significantly attenuated the vasodilatation to the same extent.These results indicate that quercetin dilates the mesenteric artery via endothelium-dependent mechanisms, and the dilatation is mainly mediated by gap junctions closely involved with endothelium-derived hyperpolarizing factor (EDHF)." @default.
• W2027166013 created "2016-06-24" @default.
• W2027166013 creator A5057165128 @default.
• W2027166013 creator A5087139089 @default.
• W2027166013 date "2013-04-01" @default.
• W2027166013 modified "2023-09-25" @default.
• W2027166013 title "Role of gap junction involved with endothelium-derived hyperpolarizing factor for the quercetin-induced vasodilatation in rat mesenteric artery" @default.
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• W2027166013 doi "https://doi.org/10.1016/j.lfs.2013.02.003" @default.
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