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• W2027704315 endingPage "436" @default.
• W2027704315 startingPage "427" @default.
• W2027704315 abstract "Increases in extracellular potassium (K + ) concentration (up to 20 mM) cause dilation in some blood vessels. This may be particularly important in myocardial ischemia because in this condition K + is released from ischemic cells. In this study, we investigated mechanisms of effect of increased K + concentration on the tone of isolated bovine coronary artery. Bovine coronary arteries were isolated and mounted in organ baths for isometric tension recording. After an equilibration period, arteries were contracted with serotonin (1 μM). When serotonin contraction reached a steady-state, K + concentration of organ baths was increased from physiological levels to 10 mM, 14 mM, 18 mM or 22 mM in four groups of the arteries. After a washout period, this procedure was repeated in presence of ouabain, a blocker of Na + /K + ATPase or a K + channel blocker (tetraethylammonium, 4-aminopyridine, glibenclamide or barium). Increasing K + concentration of the organ baths to 10 mM, 14 mM and 18 mM caused dilation in the arteries. Ouabain abolished the dilation and barium (a blocker of inward rectifier K + channels) inhibited the dilation significantly.According to our results there is K + -induced dilation in bovine coronary artery and it involves activation of both Na + /K + ATPase and inward rectifier K + channels." @default.
• W2027704315 created "2016-06-24" @default.
• W2027704315 creator A5020332948 @default.
• W2027704315 creator A5020433586 @default.
• W2027704315 date "2009-12-01" @default.
• W2027704315 modified "2023-10-14" @default.
• W2027704315 title "Potassium induced dilation in bovine coronary artery involves both inward rectifier potassium channels and Na+/K+ATPase" @default.
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• W2027704315 doi "https://doi.org/10.1556/aphysiol.96.2009.4.3" @default.
• W2027704315 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19942549" @default.
• W2027704315 hasPublicationYear "2009" @default.
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