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- W2028241988 abstract "We recently proposed a novel model of stimulation-responsive splicing for the selection of autoantigens and self-tumor antigens. Our model theorizes that the significantly higher rates of alternative splicing of autoantigen and self-tumor antigen transcripts that occur in response to stimuli could induce extra-thymic expression of untolerized antigen epitopes for elicitation of autoimmune and anti-tumor responses. To facilitate the identification of immunogenic isoforms of antigens, we have developed strategies using improved SEREX in conjunction with database-mining and immunogenic isoform mapping. Identification of immunogenic isoforms of autoantigens and self-tumor antigens is very important for the development of novel therapeutics and diagnostic tools for autoimmune diseases and tumors, such as: (1) autoantigen isoform microarrays for disease diagnosis and prognosis; (2) autoantigen isoform-specific tolerizing vaccines and splicing-redirection therapies, as well as (3) immunogenic antigen isoform-specific immunotherapy for tumors." @default.
- W2028241988 created "2016-06-24" @default.
- W2028241988 creator A5021508939 @default.
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- W2028241988 creator A5056981085 @default.
- W2028241988 creator A5069971376 @default.
- W2028241988 date "2006-11-01" @default.
- W2028241988 modified "2023-09-26" @default.
- W2028241988 title "Model of stimulation-responsive splicing and strategies in identification of immunogenic isoforms of tumor antigens and autoantigens" @default.
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