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- W2028266098 startingPage "941" @default.
- W2028266098 abstract "H elicobacter pylori is a major causative agent of gastritis and peptic ulcer disease and is an established risk factor for gastric malignancy. Antibiotic combination therapy can eradicate H . pylori . As these same regimens can evoke adverse effects and resistance, new alternative therapies or adjunctive treatments are needed. A probiotic approach may provide a novel strategy for H . pylori treatment. In the current study, two probiotic bacteria, L actobacillus acidophilus and L actobacillus reuteri, and a probiotic yeast, S accharomyces boulardii , were evaluated for their ability to influence H . pylori viability, adherence to gastric and duodenal cells, as well as the effect of S . boulardii on cell surface expression of sialic acid. Our results indicate that S . boulardii contains neuraminidase activity selective for α(2‐3)‐linked sialic acid. This neuraminidase activity removes surface α(2‐3)‐linked sialic acid, the ligand for the sialic acid‐binding H . pylori adhesin, which in turn, inhibits H . pylori adherence to duodenal epithelial cells." @default.
- W2028266098 created "2016-06-24" @default.
- W2028266098 creator A5044000016 @default.
- W2028266098 creator A5053004722 @default.
- W2028266098 date "2014-03-15" @default.
- W2028266098 modified "2023-10-02" @default.
- W2028266098 title "<i>Saccharomyces boulardii</i>expresses neuraminidase activity selective for α2,3-linked sialic acid that decreases<i>Helicobacter pylori</i>adhesion to host cells" @default.
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- W2028266098 doi "https://doi.org/10.1111/apm.12237" @default.
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