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- W2028583669 abstract "Background: In clinical practice it is far from uncommon that antihypertensive drugs fail to meet their expectations. This is mainly due to mechanisms counteracting their antihypertensive effects. These mechanisms include stimulation of the sympathetic nervous system (SNS), inhibition of the parasympathetic nervous system (PSNS), stimulation of the renin-angiotensinaldosterone system (RAAS), as well as endothelium-dependent mechanisms.Objective of study: To review whether the activation of such mechanisms follows differential patterns depending on the type of antihypertensive therapy being used.Results: The antihypertensive effects of diuretics and calcium channel blockers are largely offset by all of the mechanisms enumerated. The antihypertensive effects of ACE-inhibitors/angiotensin II(A II) receptor antagonists and beta blockers are counteracted by all of the mechanisms enumerated except for the effects of a stimulated RAAS and SNS respectively.Conclusions: ACE-inhibitors/A II receptor antagonists and beta blockers display a better profile of mechanisms counteracting their antihypertensive effects than other categories of drugs currently available." @default.
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- W2028583669 date "1998-01-01" @default.
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- W2028583669 title "Mechanisms Offsetting the Beneficial Effects of Antihypertensive Drugs: A Problem Increasingly Considered but Incompletely Understood" @default.
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