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• W2028681689 abstract "1 Balloon catheter injury to the rabbit carotid artery damaged the endothelium and induced neointima formation over 7 days. The area of intima, expressed as a percentage of the media, was 16.2 ± 4.2% and 8.2 ± 0.1% in balloon catheter-injured and sham-operated arteries. 2 Seven days after arterial injury, carotid arteries were isolated and set up as ring preparations in organ baths for isometric tension measurements. Balloon catheter-injured arteries first contracted with noradrenaline (0.01–0.1 μm), contracted further in a concentration-dependent manner to bradykinin (BK; pD2, 5.98 ± 0.22; Emax, 41.3 ± 5.2% of KCl) and to des-Arg9-BK (pD2, 7.12 ± 0.36; Emax, 46.0 ± 9.9% of KCl). In contrast, vessel segments with endothelium either intact or acutely removed were unresponsive to both BK receptor agonists. 3 The concentration-contraction curves for BK and for des-Arg9-BK were shifted to the right by the B1 receptor antagonist, [Leu8]des-Arg9-BK (3 μm), but not by the selective B2 receptor antagonist, Hoe 140 (1 μm). 4 Thus, BK and its metabolite, des-Arg9-BK act as vasoconstrictor agents following balloon catheter injury. These effects appear to be mediated by activation of B1 receptors." @default.
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• W2028681689 title "Induction of kinin B1 receptor-dependent vasoconstriction following balloon catheter injury to the rabbit carotid artery" @default.
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• W2028681689 doi "https://doi.org/10.1111/j.1476-5381.1994.tb14847.x" @default.
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