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• W2028934531 abstract "Shiga toxin‐producing Escherichia coli (STEC) is associated with hemolytic uremic syndrome (HUS). Although most clinical isolates of STEC produce hemolysin (called enterohemolysin), the precise role of enterohemolysin in the pathogenesis of STEC infections is unknown. Here we demonstrated that E. coli carrying the cloned enterohemolysin operon ( hlyC , A , B , D genes) from an STEC human strain induced the production of interleukin‐1β (IL‐1β) through its mRNA expression but not tumor necrosis factor‐α from human monocytes. No IL‐1β release was observed with an enterohemolysin (HlyA)‐negative, isogenic E. coli strain carrying a mutation in the hlyA gene. The data suggest that enterohemolysin, a pore‐forming toxin, induces the production of IL‐1β, which is one of serum risk markers for HUS." @default.
• W2028934531 created "2016-06-24" @default.
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• W2028934531 date "2002-07-12" @default.
• W2028934531 modified "2023-09-26" @default.
• W2028934531 title "Enterohemolysin operon of Shiga toxin-producing<i>Escherichia coli</i>: a virulence function of inflammatory cytokine production from human monocytes" @default.
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• W2028934531 doi "https://doi.org/10.1016/s0014-5793(02)03027-2" @default.
• W2028934531 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/12135770" @default.
• W2028934531 hasPublicationYear "2002" @default.
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