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- W2029140127 abstract "Section of one optic tract in two monkeys with a survival time of 4 months led to transsynaptic atrophy in the ipsilateral lateral geniculate nucleus. In the visual cortex (area 17) axon terminals were measured and counted and found to be slightly larger but fewer on the operated side. The same operation in 5 cats with survival times of 4–15 months again led to slightly fewer axon terminals in lamina IV of area 17, but there was no significant change of axon terminal size. The loss of terminals is unlikely to be due to retraction of optic radiation terminals, because the latter comprise only 2–5% of the axon terminals in lamina IV and a lesion of the atrophied lateral geniculate nucleus caused a number of optic radiation terminals to degenerate that was comparable with the normal side. The reduction in the number of axon terminals in area 17 was thus a secondary effect on axon terminals whose cell bodies probably lay within the cortex. In contrast to area 17, deprived of neural input by tract section, a part of area 18 receiving a callosal projection from the contralateral hemisphere was studied in the two tract-sectioned monkeys. In this area there was a slightly greater density of axon terminals on the tract-sectioned side than on the control side. The meaning of of these results is discussed." @default.
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- W2029140127 date "1971-11-01" @default.
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- W2029140127 title "The fate of axon terminals in visual cortex during transsynaptic atrophy of the lateral geniculate nucleus" @default.
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- W2029140127 doi "https://doi.org/10.1016/0006-8993(71)90350-7" @default.
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