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- W2029276498 abstract "This study was performed to investigate whether nitric oxide (NO) precursor (<i>L</i>-arginine), NO donor (S-nitroso-N-acetylpenicillamine, SNAP) and NO synthase inhibitors [N<sup>G</sup>-nitro-<i>L</i>-arginine-methylester (<i>L</i>-NAME) and N<sup>G</sup>-nitro-<i>L</i>-arginine (<i>L</i>-NOARG)] modulate morphine-induced anxiolytic effects in the plus-maze. <i>L</i>-Arginine (100, 200 and 300 mg kg<sup>–1</sup>, i.p.) and SNAP (4, 8 and 10 mg kg<sup>–1</sup>, i.p.) reduced the anxiolytic effect of morphine (20 mg kg<sup>–1</sup>, s.c.). <i>L</i>-NAME (10, 20 and 40 mg/kg, i.p.) and <i>L</i>-NOARG (10, 15 and 20 mg kg<sup>–1</sup>, i.p.) enhanced the anxiolytic effects of morphine (20 mg kg<sup>–1</sup>, s.c.). On the other hand, <i>L</i>-arginine and SNAP increased the morphine-induced locomotor activity. <i>L</i>-NAME decreased the morphine-induced locomotor activity, but <i>L</i>-NOARG did not modify the morphine-induced locomotor activity. Therefore, these results suggest that the anxiolytic effects of morphine can be modulated by NO systems." @default.
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- W2029276498 date "2003-01-01" @default.
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- W2029276498 title "Anxiolytic Effects of Acute Morphine Can Be Modulated by Nitric Oxide Systems" @default.
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- W2029276498 doi "https://doi.org/10.1159/000070457" @default.
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