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• W2029327504 abstract "Abstract The c-Myb transcription factor controls differentiation and proliferation in hematopoietic and other cell types and has latent transforming activity, but little is known about its regulation during the cell cycle. Here, c-Myb was identified as part of a protein complex from human T cells containing the cyclin-dependent kinase (CDK) CDK6. Assays using model reporter constructs as well as endogenous target genes showed that the activity of c-Myb was inhibited by cyclin D1 plus CDK4 or CDK6 but stimulated by expression of the CDK inhibitors p16 Ink4a, p21 Cip1, or p27 Kip1. Mapping experiments identified a highly conserved region in c-Myb which, when transferred to the related A-Myb transcription factor, also rendered it responsive to CDKs and p27. The results suggest that c-Myb activity is directly regulated by cyclin D1 and CDKs and imply that c-Myb activity is regulated during the cell cycle in hematopoietic cells." @default.
• W2029327504 created "2016-06-24" @default.
• W2029327504 creator A5027429714 @default.
• W2029327504 creator A5033073898 @default.
• W2029327504 creator A5069563017 @default.
• W2029327504 date "2005-05-15" @default.
• W2029327504 modified "2023-09-27" @default.
• W2029327504 title "Positive and negative regulation of c-Myb by cyclin D1, cyclin-dependent kinases, and p27 Kip1" @default.
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• W2029327504 doi "https://doi.org/10.1182/blood-2004-08-3342" @default.
• W2029327504 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1895079" @default.
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