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- W2029397966 endingPage "15" @default.
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- W2029397966 abstract "Increasing evidence from epidemiological, preclinical and clinical studies suggests that dysregulated inflammatory response plays a pivotal role in a multitude of chronic ailments including cancer. The molecular mechanism(s) by which chronic inflammation drives cancer initiation and promotion include increased production of pro-inflammatory mediators, such as cytokines, chemokines, reactive oxygen intermediates, increased expression of oncogenes, COX-2 (cyclo-oxygenase-2), 5-LOX (5-lipoxygenase) and MMPs (matrix metalloproteinases), and pro-inflammatory transcription factors such as NF-κB (nuclear factor κB), STAT3 (signal transducer and activator of transcription 3), AP-1 (activator protein 1) and HIF-1α (hypoxia-inducible factor 1α) that mediate tumour cell proliferation, transformation, metastasis, survival, invasion, angiogenesis, chemoresistance and radioresistance. These inflammation-associated molecules are activated by a number of environmental and lifestyle-related factors including infectious agents, tobacco, stress, diet, obesity and alcohol, which together are thought to drive as much as 90% of all cancers. The present review will focus primarily on the role of various inflammatory intermediates responsible for tumour initiation and progression, and discuss in detail the critical link between inflammation and cancer." @default.
- W2029397966 created "2016-06-24" @default.
- W2029397966 creator A5002262324 @default.
- W2029397966 creator A5034304968 @default.
- W2029397966 creator A5051799874 @default.
- W2029397966 creator A5058193427 @default.
- W2029397966 creator A5060581224 @default.
- W2029397966 date "2011-09-26" @default.
- W2029397966 modified "2023-10-10" @default.
- W2029397966 title "Multifaceted link between cancer and inflammation" @default.
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