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• W2029453023 startingPage "199" @default.
• W2029453023 abstract "Focal, freeze-induced lesions were made in isolated hemispheres of turtle cerebral cortex in vitro, permitting the investigation of epileptiform discharges in a preparation with preserved intracortical circuitry. Freeze lesions resulted in interictal discharges and occasional ictal-like events. The interictal discharges were dependent upon activation of non-NMDA excitatory amino acid receptors and were affected by but did not require NMDA receptor activation. Voltage clamp and current clamp recordings revealed abnormal bursts of low-amplitude action potentials in 36% of recorded neurons, while large, repetitive inhibitory potentials, mediated by GABAA receptors, were recorded in 90% of the neurons. Thus, prominent findings in this model include abnormalities of both excitatory and inhibitory activity. Since these changes in neuronal excitability resulted from a localized physical injury, they may resemble the changes that occur in acute posttraumatic epilepsy." @default.
• W2029453023 created "2016-06-24" @default.
• W2029453023 creator A5015724734 @default.
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• W2029453023 date "1992-03-01" @default.
• W2029453023 modified "2023-10-12" @default.
• W2029453023 title "Abnormal Action-Potential Bursts and Synchronized, GABA-Mediated Inhibitory Potentials in an In Vitro Model of Focal Epilepsy" @default.
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• W2029453023 doi "https://doi.org/10.1111/j.1528-1157.1992.tb02308.x" @default.
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