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• W2029488286 abstract "We recently reported that the nonclassical HLA class I molecule HLA-G was expressed in the endomyocardial biopsies and sera of 16% of heart transplant patients studied. The aim of the present report is to identify cells that may be responsible for HLA-G protein expression during the allogeneic reaction. Carrying out mixed lymphocyte cultures in which the responder cell population was depleted either in CD4 + or CD8 + T cells, we found that soluble HLA-G5 protein but not the membrane-bound HLA-G isoform was secreted by allo-specific CD4 + T cells from the responder population, which suppressed the allogeneic proliferative T cell response. This inhibition may be reversed by adding the anti-HLA-G 87G antibody to a mixed lymphocyte culture. That may indicate a previously uncharacterized regulatory mechanism of CD4 + T cell proliferative response." @default.
• W2029488286 created "2016-06-24" @default.
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• W2029488286 date "2001-09-25" @default.
• W2029488286 modified "2023-10-16" @default.
• W2029488286 title "Soluble HLA-G protein secreted by allo-specific CD4⁺T cells suppresses the allo-proliferative response: A CD4⁺T cell regulatory mechanism" @default.
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• W2029488286 doi "https://doi.org/10.1073/pnas.201407398" @default.
• W2029488286 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/59783" @default.
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