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• W2029551841 endingPage "1924" @default.
• W2029551841 startingPage "1917" @default.
• W2029551841 abstract "Adenosine receptor agonists and agents enhancing pericellular concentrations of adenosine possess antiinflammatory properties. In the present study, we found that R-phenylisopropyladenosine (R-PIA), 5'-N-ethylcarboxamido adenosine (NECA), other agonists of adenosine receptors and dipyridamole, an adenosine uptake inhibitor, inhibited tumor necrosis factor (TNF) production by endotoxin-stimulated human monocytes in a concentration-dependent manner with no inhibition of interleukin-6. The rank order of agonist potency is characteristic of neither A1 nor A2 receptors and suggests the involvement of another receptor subtype. The effect of R-PIA on TNF was in part abolished by the antagonist 8-sulfophenyltheophylline. In endotoxin-treated rats, R-PIA pretreatment (2.5 mg/kg) reduced serum TNF levels by 98%, with no modification of serum IL6 levels. TNF inhibition could be an important mechanism by which adenosine analogs exert their antiinflammatory action." @default.
• W2029551841 created "2016-06-24" @default.
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• W2029551841 date "1993-01-01" @default.
• W2029551841 modified "2023-10-01" @default.
• W2029551841 title "Inhibition of human monocyte TNF production by adenosine receptor agonists" @default.
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• W2029551841 doi "https://doi.org/10.1016/0024-3205(93)90632-d" @default.
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