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- W2029708529 abstract "Summary Pretreatment of mice with cyclophosphamide (CY) increases susceptibility of adult mice to herpes simplex type I (HSV-I) infection. In searching for a CY sensitive mechanism, our attention has been focused on natural killer (NK) cells. Activity of these cells was increased in spleens of adult mice after inoculation with HSV-I and depressed after CY administration. The timing of recovery of NK cell activity after the administration of CY, was consistent with a role for these cells in collaboration with immune T lymphocytes when transferred to pre-treated, virus infected animals. It is postulated that sensitized T lymphocytes reacting with virus antigens release products that activate macrophages which in turn produce interferon. Interferon increases NK cell activity and NK cells inhibit viral replication. A correlation between resistance to HSV-I infection and NK cell activity of CBA or C57BL/10 mice was found. It is suggested that NK cells play a role in defence, not only against tumour cells, but also against a wide range of infective agents." @default.
- W2029708529 created "2016-06-24" @default.
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- W2029708529 date "1980-03-01" @default.
- W2029708529 modified "2023-09-27" @default.
- W2029708529 title "The role of natural killer (NK) cells in resistance to herpes simplex virus I (HSV-I) infection" @default.
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- W2029708529 doi "https://doi.org/10.1111/j.1365-3024.1980.tb00044.x" @default.
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