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- W2029772003 abstract "Within less than a year after its epidemic started (in December 2013) in Guinea, Ebola virus (EBOV), a member of the filoviridae, has spread over a number of West-African countries (Guinea, Sierra Leone and Liberia) and gained allures that have been unprecedented except by human immunodeficiency virus (HIV). Although EBOV is highly contagious and transmitted by direct contact with body fluids, it could be counteracted by the adequate chemoprophylactic and -therapeutic interventions: vaccines, antibodies, siRNAs (small interfering RNAs), interferons and chemical substances, i.e. neplanocin A derivatives (i.e. 3-deazaneplanocin A), BCX4430, favipiravir (T-705), endoplasmic reticulum (ER) α-glucosidase inhibitors and a variety of compounds that have been found to inhibit EBOV infection blocking viral entry or by a mode of action that still has to be resolved. Much has to be learned from the mechanism of action of the compounds active against VSV (vesicular stomatitis virus), a virus belonging to the rhabdoviridae, that in its mode of replication could be exemplary for the replication of filoviridae." @default.
- W2029772003 created "2016-06-24" @default.
- W2029772003 creator A5040662398 @default.
- W2029772003 date "2015-01-01" @default.
- W2029772003 modified "2023-10-09" @default.
- W2029772003 title "Ebola virus (EBOV) infection: Therapeutic strategies" @default.
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- W2029772003 doi "https://doi.org/10.1016/j.bcp.2014.11.008" @default.
- W2029772003 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7110990" @default.
- W2029772003 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25481298" @default.
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