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• W2029958219 abstract "Leukocyte interactions with vascular endothelium are an initial step for leukocyte entry into infectious foci where endothelial selectins may play a key role. Infiltrating leukocyte is essential for bacterial clearance, suggesting that endothelial selectins would be important in host defense against microorganisms. To address this, E-, P-, and E/P-selectin-deficient mice (E−/−, P−/−, E/P−/−) and wild-type (WT) mice underwent cecal ligation and puncture (CLP). Neither leukocyte infiltration nor bacterial load in the peritoneum was altered in E−/−, P−/−, and E/P−/− mice compared to WT mice. However, E−/−, P−/−, and E/P−/− mice were resistant to the lethality induced by CLP. At the mechanistic level, E−/−, P−/−, and E/P−/− mice did not develop renal dysfunction, a possible cause of death during sepsis. The serum level of interleukin-13 in E−/−, P−/−, and E/P−/− mice that had undergone CLP was higher than that in WT mice, whereas levels of macrophage inflammatory protein-2, KC in serum, and KC in kidney were lower than those in WT mice. These experiments demonstrate that endothelial selectin-mediated leukocyte rolling is not required for leukocyte entry in septic peritonitis and that endothelial selectins may affect mice survival during sepsis by influencing the cytokine profiles." @default.
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• W2029958219 date "2002-02-01" @default.
• W2029958219 modified "2023-09-26" @default.
• W2029958219 title "Mice Genetically Lacking Endothelial Selectins Are Resistant to the Lethality in Septic Peritonitis" @default.
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• W2029958219 doi "https://doi.org/10.1006/exmp.2001.2416" @default.
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