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- W2030184490 abstract "The aim of the present study is to determine the role of intracellular Ca 2+ in VEGF signaling. We demonstrate that reduction in Ca 2+ by chelating compound BAPTA‐AM or by IP 3 ‐endoplasmic reticulum blocker 2‐APB selectively inhibited VEGF‐induced activation of c‐Src‐PI3K‐Akt but not ERK1/2 in human coronary artery endothelial cells (HCAEC). We also show that the selective inhibitory effects of NADPH oxidase knockdown on VEGF‐mediated activation of c‐Src‐PI3K‐Akt signaling and cell proliferation in HCAEC can be reversed by increase in intracellular Ca 2+ . These results suggest an essential role for Ca 2+ in redox‐dependent selective activation of c‐Src‐PI3K‐Akt and endothelial cell proliferation." @default.
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- W2030184490 date "2010-06-01" @default.
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- W2030184490 title "Intracellular Ca2+ can compensate for the lack of NADPH oxidase-derived ROS in endothelial cells" @default.
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- W2030184490 doi "https://doi.org/10.1016/j.febslet.2010.05.053" @default.
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