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- W2030275496 abstract "The effect of APC loss on azoxymethane (AOM)-induced apoptosis and cell proliferation, as well as their regulation by sulindac was examined in colon and small intestine in APC(Min/+) mice. APC(Min/+) mice showed increased epithelial proliferation in all regions, with significant impairment of apoptosis in small intestine, but not in colon. Sulindac administration restored defective apoptosis to normal. As the apoptotic defect occurred at the major site of intestinal tumor formation in APC(Min/+) mice and as it was restored to normal by a proven chemopreventive agent, this defect in apoptosis might be a key biological consequence of APC dysfunction contributing to tumor formation." @default.
- W2030275496 created "2016-06-24" @default.
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- W2030275496 date "2007-04-01" @default.
- W2030275496 modified "2023-09-27" @default.
- W2030275496 title "Defective acute apoptotic response to genotoxic carcinogen in small intestine of APCMin/+ mice is restored by sulindac" @default.
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- W2030275496 doi "https://doi.org/10.1016/j.canlet.2006.07.009" @default.
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