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• W2030338002 abstract "1 In conscious fasted rabbits an intravenous infusion of clonidine (2 μg kg−1 min−1) induced hyperglycaemia. The increase in blood glucose was accompanied by an inhibition of insulin secretion and basal lipolysis. 2 Yohimbine infused at a rate of 20 μg kg−1 min−1 suppressed clonidine-induced hyperglycaemia and blocked the inhibitory effect on insulin secretion mediated by the α2-adrenoceptor agonist. 3 The intravenous infusion of amidephrine (10 μg kg−1 min−1) induced an increase in insulin secretion in the absence of patent hyperglycaemia. Prazosin, 0.3 mg kg−1 s.c. selectively antagonized the effect of amidephrine on insulin secretion. 4 Isoprenaline infusion (4.4 μg kg−1 min−1) evoked a significant increase in blood glycerol and immunoreactive insulin plasma levels. Both responses were clearly attenuated when α2-adrenoceptors were simultaneously stimulated by selective (clonidine) and less selective (phenylephrine, 20 μg kg−1 min−1) agonists. 5 Amidephrine infusion did not induce appreciable changes in blood glycerol nor did it modify, isoprenaline-induced lipolytic response. 6 Simultaneous infusion of isoprenaline and amidephrine induced a remarkable increase in insulin secretion. 7 It is concluded that in normal fasted rabbits stimulation of α2-adrenoceptors depresses basal and β-adrenoceptor mediated lipolysis and insulin secretion. On the other hand, selective stimulation of α1-adrenoceptors does not affect lipolysis but induces insulin release. Simultaneous stimulation of α1- and β-adrenoceptors potentiates the insulin secretory response." @default.
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• W2030338002 date "1988-06-01" @default.
• W2030338002 modified "2023-10-17" @default.
• W2030338002 title "Role of α1- and α2-adrenoceptors in catecholamine-induced hyperglycaemia, lipolysis and insulin secretion in conscious fasted rabbits" @default.
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• W2030338002 doi "https://doi.org/10.1111/j.1476-5381.1988.tb11531.x" @default.
• W2030338002 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1853986" @default.
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