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• W2030481178 abstract "AraC resistance in vitro is explained by inactivation of dCK, while resistance to DNR is described by overexpression of multidrug efflux pumps like Pgp or MRP. Thusfar, no correlation between resistance mechanisms in vitro and in patients with AML has been documented. We generated AraC and DNR double resistant cell lines to investigate resistance mechanisms of both agents. In these cell lines involvement of dCK was extensively investigated and Pgp expression and activity was determined. Our data implicate that similar resistance mechanisms like inactivation of dCK coincided by alternatively spliced dCK forms and overexpression of Pgp are induced in single—as well as in double resistant leukemic cell lines." @default.
• W2030481178 created "2016-06-24" @default.
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• W2030481178 date "2003-05-01" @default.
• W2030481178 modified "2023-09-26" @default.
• W2030481178 title "Inactivation of deoxycytidine kinase and overexpression of P-glycoprotein in AraC and daunorubicin double resistant leukemic cell lines" @default.
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• W2030481178 doi "https://doi.org/10.1016/s0145-2126(02)00224-2" @default.
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• W2030481178 hasPublicationYear "2003" @default.
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