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- W2030589675 abstract "Chronic inflammation predisposes patients with inflammatory bowel disease to the risk of developing colitis-associated cancer (CAC). Growing evidence strongly suggests that CAC development is multifactorial and is attributed to concurrent, dynamic dysregulations in host immunity, enteric microbiota, and epithelial restitution during the course of chronic inflammation. This article discusses the recent advances in understanding the different forms of CAC that may develop in patients with inflammatory bowel disease and animal models, as well as molecular alterations and other processes that orchestrate the development of CAC." @default.
- W2030589675 created "2016-06-24" @default.
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- W2030589675 date "2014-11-01" @default.
- W2030589675 modified "2023-10-18" @default.
- W2030589675 title "Recent Advancement in Understanding Colitis-associated Tumorigenesis" @default.
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- W2030589675 doi "https://doi.org/10.1097/mib.0000000000000094" @default.
- W2030589675 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4206829" @default.
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