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- W2030795831 abstract "Glucocorticoids enhance β-adrenoceptor (βAR) coupling to GS protein, increase the stability of steady state levels of βAR mRNA and enhance membrane expression of βAR. Glucocorticoids enhance the expression of GS protein. Hypercortisolemia and escape from the dexamethasone suppression test were reported in a subgroup of depressed patients. Imipramine has been shown to reduce the phosphorylation of the cyclase repsponse element-binding protein and consequently the transcriptional activity of the cyclase response element. We recently reported increased pretreatment β-adrenoceptor density and coupling in tricyclic antidepressant-responsive patients only, while βAR function was normal in non-responsive patients. Downregulation and uncoupling of βAR was seen in imipramine-responsive patients, but βAR remained unchanged in treatment non-responsive patients." @default.
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- W2030795831 date "2000-04-01" @default.
- W2030795831 modified "2023-09-26" @default.
- W2030795831 title "285. Effects of hydrocortisone on β-adrenoceptor coupling to GS protein in depression before and after treatment" @default.
- W2030795831 doi "https://doi.org/10.1016/s0006-3223(00)00549-7" @default.
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