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• W2031034568 abstract "Epileptic seizures represent dysfunctional neural networks dominated by excessive and/or hypersynchronous activity. Recent progress in the field has outlined two concepts regarding mechanisms of seizure generation, or ictogenesis. First, all seizures, even those associated with what have historically been thought of as 'primary generalized' epilepsies, appear to originate in local microcircuits and then propagate from that initial ictogenic zone. Second, seizures propagate through cerebral networks and engage microcircuits in distal nodes, a process that can be weakened or even interrupted by suppressing activity in such nodes. We describe various microcircuit motifs, with a special emphasis on one that has been broadly implicated in several epilepsies: feed-forward inhibition. Furthermore, we discuss how, in the dynamic network in which seizures propagate, focusing on circuit 'choke points' remote from the initiation site might be as important as that of the initial dysfunction, the seizure 'focus'." @default.
• W2031034568 created "2016-06-24" @default.
• W2031034568 creator A5033400284 @default.
• W2031034568 creator A5076444204 @default.
• W2031034568 date "2015-02-24" @default.
• W2031034568 modified "2023-10-01" @default.
• W2031034568 title "Microcircuits and their interactions in epilepsy: is the focus out of focus?" @default.
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