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• W2031539368 abstract "A rabbit eye model of neural ischaemia is described that uses an increased pressure in the anterior eye chamber to block the capillary supply to the retina. A microdialysis probe placed very close to the retinal surface was used to monitor release of amino acids during ischaemia. A large (two- to threefold) increase in the release of glutamate and O-phosphoserine (twofold), but not of six other amino acids monitored, occurred during initial ischaemia. During reperfusion after release of intraocular pressure, much larger (five- to 10-fold) increases in the release of these amino acids were observed. Parallel ischaemic retinal tissue damage was observed. This damage was prevented by ketamine applied locally via a superfusion needle, suggesting that glutamate released during ischaemia, and particularly during reperfusion, was responsible for cell death." @default.
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• W2031539368 date "1992-07-01" @default.
• W2031539368 modified "2023-10-14" @default.
• W2031539368 title "Glutamate Release in Experimental Ischaemia of the Retina: An Approach Using Microdialysis" @default.
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• W2031539368 doi "https://doi.org/10.1111/j.1471-4159.1992.tb08912.x" @default.
• W2031539368 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/1351929" @default.
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