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- W2031663138 abstract "The response to ACTH1–39 of adrenal cells from fetal sheep, monkeys and guinea pigs were compared with that of cells from adult glands. Although the cortisol production rate was lower from the fetal cells they responded to physiological rises in ACTH concentration by large increases in output. The proportional increase in cortisol production was of the same order in the fetal and adult tissue. The in vitro responses of fetal tissue contrasts with the data available on in vivo function. Analysis of ACTH in fetal plasma and pituitary show it to be present in high-molecular-weight peptides and ACTH1–39 At the time of adrenal hypertrophy and increase cortisol secretion prior to birth the concentration ratio in plasma of ACTH1–39/“big” peptides increases markedly. The big peptides inhibit the response to ACTH1–39 of the fetal adrenal cells. These peptides may be responsible for suppressing the fetal adrenal in vivo and the fall in their relative contribution, late in gestation, to the total ACTH pool may be one of the factors initiating parturition." @default.
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- W2031663138 date "1980-01-01" @default.
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- W2031663138 title "Acth peptides and the development of the fetal adrenal" @default.
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- W2031663138 doi "https://doi.org/10.1016/0022-4731(80)90253-8" @default.
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