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- W2031738356 abstract "The effects of methimazole or betamethasone therapy on the TSH receptor antibody response to radioiodine therapy were compared in a prospective randomized study of 60 patients with hyperthyroidism due to Graves' disease. The patients were followed for 1 yr after treatment with 131I. Twentythree patients received 131I alone, 17 were treated with methimazole for 2 months before and 3 months after 131I therapy, and 20 patientswere treated with betamethasone for 3 weeks before and 4 weeks after 131I therapy. 131I induced a transient rise in the meanserum level of TSH receptor autoantibodies, measured as TSH; binding inhibitory immunoglobulin (TBII), but in patients receiving methimazole treatment, no such rise occurred. In the betamethasone-treated patients, TBII increased similarly to that in patients treated with 131Ialone. In addition, in patients given betamethasone, there was an early decrease in total serum immunoglobulin G, which persisted throughout the follow-up period. In the other 2 groups, no changes in total immunoglobulin G were found. The results demonstrate that in hyperthyroid Graves' disease, TBII, production is influenced by therapy. Methimazole abolished the 131I-induced increase in TBII, whereas betamethasone did not have such an inhibitory effect." @default.
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- W2031738356 date "1986-04-01" @default.
- W2031738356 modified "2023-10-14" @default.
- W2031738356 title "Methimazole, but not Betamethasone, Prevents<sup>131</sup>I Treatment-Induced Rises in Thyrotropin Receptor Autoantibodies in Hyperthyroid Graves' Disease*" @default.
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- W2031738356 doi "https://doi.org/10.1210/jcem-62-4-773" @default.
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