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- W2032266893 abstract "The effects of prostaglandin E 2 (PGE 2 ) and of several synthetic prostanoids on the cardiac response to sympathetic nerve stimulation in guinea‐pig atria have been evaluated. PGE 2 (0.01–100 n m ), sulprostone (0.01–100 n m ) and misoprostol (0.1–100 n m ), but not butaprost (0.1–100 n m ), dose‐dependently reduced the increase in cardiac contractility induced by electrical field stimulation of sympathetic terminals. The EP 1 antagonist AH6809 (1 μ m ) did not modify the inhibition of cardiac response induced by PGE 2 , sulprostone and misoprostol. In preparations preloaded with [ 3 H]‐noradrenaline, tritium overflow induced by electrical field stimulation was greatly and significantly reduced by 100 n m PGE 2 and by 100 n m sulprostone. These results indicate that PGE 2 and other synthetic prostanoids reduce noradrenaline release from cardiac adrenergic nerve terminals acting on prejunctional inhibitory receptors belonging to the EP 3 subtype." @default.
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- W2032266893 date "1991-03-01" @default.
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- W2032266893 title "Prejunctional prostanoid receptors on cardiac adrenergic terminals belong to the EP3 subtype" @default.
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- W2032266893 doi "https://doi.org/10.1111/j.1476-5381.1991.tb12214.x" @default.
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