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- W2033533185 abstract "The mutagenic activity present in urine of animals treated with acrylonitrile (ACN) is tentatively related to the excretion of three urinary metabolites: thiocyanate (SCN−), hydroxyethylmercapturic acid (OH-MA), and cyanoethylmercapturic acid (CN-MA). It is shown that the route of administration and animal species affect SCN− excretion but not the excretion of hydroxyethyl- and cyanoethylmercapturic acids or the mutagenicity of urine from ACN-treated animals. Various pretreatments (phenobarbital, CoCl2, diethylmaleate, trichloroacetonitrile) decrease the mutagenicity of urine from ACN-treated animals and decrease the excretion of SCN− and OH-MA. None of the quantified urinary metabolites is responsible for urinary mutagenicity." @default.
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- W2033533185 title "Metabolism and mutagenicity of acrylonitrile: An in vivo study" @default.
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- W2033533185 doi "https://doi.org/10.1002/em.2860070505" @default.
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